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anti cleaved full length caspase 3  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc anti cleaved full length caspase 3
    Anti Cleaved Full Length Caspase 3, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 42345 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/length+cleaved+caspase+3/Caspase-3+Antibody/pmc10929186-132-28-31
    Average 99 stars, based on 42345 article reviews
    anti cleaved full length caspase 3 - by Bioz Stars, 2026-10
    99/100 stars

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    Western Blot:

    Article Title: Inhibition of Aβ(25-35)-induced cell apoptosis by low-power-laser-irradiation (LPLI) through promoting Akt-dependent YAP cytoplasmic translocation.
    Article Snippet: Contents lists available at SciVerse ScienceDirect Cellular Signalling j ourna l homepage: www.e lsev ie r .com/ locate /ce l l s ig Inhibition of Aβ25–35-induced cell apoptosis by Low-power-laser-irradiation (LPLI) through promoting Akt-dependent YAP cytoplasmic translocation Heng Zhang, Shengnan Wu, Da Xing ⁎ MOE Key Laboratory of Laser Life Science & Institute of Laser Life Science, College of Biophotonics, South China Normal University, Guangzhou 510631, China ⁎ Corresponding author.. Tel.. : +86 20 85210089; fax: E-mail address: xingda@scnu.edu.cn (D. Xing).

    Article Title: YAP accelerates Aβ(25-35)-induced apoptosis through upregulation of Bax expression by interaction with p73.
    Article Snippet: Accumulation of amyloid-b-peptide (Ab) in the brain is considered as a pathological hallmark of Alzheimer’s disease (AD).. Previous studies show that p73 is vital for mediating the pathogenic process of AD.. Yes-associated protein (YAP) has been shown to positively regulate p73 in promoting apoptosis induced by anti-cancer agents.



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    length cleaved caspase 3  (Cell Signaling Technology Inc)
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    Pharmacodynamic analysis of eribulin, irinotecan and the combination in ES-4 xenografts. (A) Immunoblotting of three independent ES-4 tumor lysates for markers of mitotic arrest (P-S10-histone H3), DNA damage, (P-S139-H2A.X; P-S345-Chk1; P-T68-Chk2; RRM2B, ribonucleotide reductase regulatory TP53-inducible subunit M2B), TP53-regulated genes (TP53; cyclin dependent kinase inhibitor 1A, p21; MDM2; PUMA) and apoptosis (full length and cleaved caspase 3; cleaved PARP). GAPDH was used as a loading control. (B) Immunohistochemistry for TP53 in ES-4 xenografts treated with eribulin, irinotecan or the combination. Tumors were excised and processed at 24h and 144h after the first drug dose. (C) Quantification of TP53 staining in ES-4 xenografts. *p < 0.033, **p < 0.002, ***p < 0.001 compared to control; #p < 0.033 compared to eribulin + irinotecan at 24 h; $p < 0.033, $$p < 0.002 compared to eribulin + irinotecan at 144h, two-way ANOVA with Tukey’s post-hoc test. ERB, eribulin; IRN, irinotecan; ERB+IRN, combination treatment.
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    Pharmacodynamic analysis of eribulin, irinotecan and the combination in ES-4 xenografts. (A) Immunoblotting of three independent ES-4 tumor lysates for markers of mitotic arrest (P-S10-histone H3), DNA damage, (P-S139-H2A.X; P-S345-Chk1; P-T68-Chk2; RRM2B, ribonucleotide reductase regulatory TP53-inducible subunit M2B), TP53-regulated genes (TP53; cyclin dependent kinase inhibitor 1A, p21; MDM2; PUMA) and apoptosis (full length and cleaved caspase 3; cleaved PARP). GAPDH was used as a loading control. (B) Immunohistochemistry for TP53 in ES-4 xenografts treated with eribulin, irinotecan or the combination. Tumors were excised and processed at 24h and 144h after the first drug dose. (C) Quantification of TP53 staining in ES-4 xenografts. *p < 0.033, **p < 0.002, ***p < 0.001 compared to control; #p < 0.033 compared to eribulin + irinotecan at 24 h; $p < 0.033, $$p < 0.002 compared to eribulin + irinotecan at 144h, two-way ANOVA with Tukey’s post-hoc test. ERB, eribulin; IRN, irinotecan; ERB+IRN, combination treatment.

    Journal: Clinical cancer research : an official journal of the American Association for Cancer Research

    Article Title: Evaluation of Eribulin Combined with Irinotecan for Treatment of Pediatric Cancer Xenografts.

    doi: 10.1158/1078-0432.CCR-19-1822

    Figure Lengend Snippet: Pharmacodynamic analysis of eribulin, irinotecan and the combination in ES-4 xenografts. (A) Immunoblotting of three independent ES-4 tumor lysates for markers of mitotic arrest (P-S10-histone H3), DNA damage, (P-S139-H2A.X; P-S345-Chk1; P-T68-Chk2; RRM2B, ribonucleotide reductase regulatory TP53-inducible subunit M2B), TP53-regulated genes (TP53; cyclin dependent kinase inhibitor 1A, p21; MDM2; PUMA) and apoptosis (full length and cleaved caspase 3; cleaved PARP). GAPDH was used as a loading control. (B) Immunohistochemistry for TP53 in ES-4 xenografts treated with eribulin, irinotecan or the combination. Tumors were excised and processed at 24h and 144h after the first drug dose. (C) Quantification of TP53 staining in ES-4 xenografts. *p < 0.033, **p < 0.002, ***p < 0.001 compared to control; #p < 0.033 compared to eribulin + irinotecan at 24 h; $p < 0.033, $$p < 0.002 compared to eribulin + irinotecan at 144h, two-way ANOVA with Tukey’s post-hoc test. ERB, eribulin; IRN, irinotecan; ERB+IRN, combination treatment.

    Article Snippet: Proteins were transferred to Immobilon-FL PVDF membranes (MilliporeSigma) and probed with antibodies for cleaved PARP (Cell Signaling #5625), phospho-S139-H2A.X (#9718), TP53 (#48818), CDKN1A/p21 (#2947), MDM2 (#86934), PUMA/BBC3 (#12450) phospho-S345-Chk1 (#2348), Chk1 (#2360), phospho-T68-Chk2 (#2197), Chk2 (#3440), full length/cleaved caspase 3 (#9668), cleaved caspase 3 (#9664), phospho-S10-histone H3 (#53348), histone H3 (#4499), GAPDH (#2118) or RRM2B/p53R2 (Invitrogen #PA5–19970).

    Techniques: Western Blot, Immunohistochemistry, Staining